A paper published in the journal Proceedings of the National Academy of Science on Monday reports that the bacterium Akkermansia muciniphila, which is normally present in the gut lining and which increases the thickness of the mucus layer of the gut, shows reduced concentration in obese and type-2 diabetic mice; additionally, feeding these mice with live A. muciniphila reversed the high-fat-diet induced conditions, including type-2 diabetes, fat-mass gain, and metabolic endotoxemia. Administration of heat-killed A. muciniphila had none of these effects, showing that the live bacteria are responsible for the effects.

Abstract: http://www.pnas.org/content/early/2013/05/08/1219451110
The full text is available for free.

The obesity epidemic in mice is over!

:P

Bewdy!

So how long before it gets added to some form of yoghurt?

Yeah but the mice probably fart more and poop their undies.

So how do these bacteria actually work?

Bubblecar said:

So how do these bacteria actually work?

>However, the precise physiological roles played by this bacterium during obesity and metabolic disorders are unknown.

OK. I still want a load of these germs in my system, and I want them now!

AussieDJ said:

Bewdy!

So how long before it gets added to some form of yoghurt?

and mcburgers and dairy foods and all those fast foods, junk foods etc

http://www.the-scientist.com/?articles.view/articleNo/34346/title/Do-Mice-Make-Bad-Models-/

Biomedical scientists have long relied on experimentation in mice to explore human disease and evaluate drug candidates. But mouse models do not accurately reflect the genetic and proteomic responses to acute inflammatory stress in humans, according to a new study.