Rotavirus can speed up diabetes onset
see article
http://www.abc.net.au/science/articles/2014/03/28/3971982.htm
This seems like another good reason to support opting for this vaccine for rotavirus
Rotavirus can speed up diabetes onset
see article
http://www.abc.net.au/science/articles/2014/03/28/3971982.htm
This seems like another good reason to support opting for this vaccine for rotavirus
Makes me wonder if there is an evolutionary advantage to the rotavirus for altering the body’s capacity to control the sugar levels.
A little bit like a germ or virus that was discovered to inhabit the junk DNA of an insect and control the gender of the next generation. This caused an evolutionary advantage to the virus/germ.
I don’t have a link but GeoffD posted that thread on our beloved SSSF. The title resembled something similar to re-think Junk DNA or something like that.
There’s a couple of old ones here.
It was discussed quite a bit back then but most of the links are 404s
http://www2b.abc.net.au/science/k2/stn/archives/archive34/default.shtm
http://www2.abc.net.au/science/k2/stn/january2000/posts/topic22969.shtm
I wasn’t aware of this avatar pwm “Dr. Ed G”
http://en.wikipedia.org/wiki/Rotavirus
Enteroviruses apparently have been under suspicion for a while, but note one study elsewhere shows infection of mice with lymphocytic choriomeningitis virus stops the development.
This one suggests too things can go either way.
http://diabetes.diabetesjournals.org/content/57/11/2863.full
“In humans, the prime candidates for infectious causes of type 1 diabetes are enteroviruses such as CVB. In mice, and most notably in NOD mice, CVB also appears associated with the development of autoimmunity. Early studies have shown that infection of normal mice with CVB4 causes a diabetic state associated with low insulin levels consistent with islet cell destruction (33). CVB4 has since been shown to be tightly associated with the initiation of type 1 diabetes in the NOD mouse. However, the influence of the virus appears to be contingent upon the precise point in time at which infection occurs (34). The B3 strain of CVB3, in contrast, mediates significant protection against type 1 diabetes development in NOD mice regardless of the time of infection”
I wonder what the ideal conditions are for the rotavirus to thrive are, for example. Virus cells need nutrition too.. yeah?